Immagine di GAIA CODOLO

GAIA CODOLO

Title: Ricercatore a tempo determinato di tipo A

SSD: MED/04 - General Pathology

Address: VIA U. BASSI, 58/B - PADOVA

Phone: 0498276308

E-mail: gaia.codolo@unipd.it

Teaching in current academic year

Course Degree
IMMUNOLOGYMolecular biology

Curriculum

Gaia Codolo Assistant Professor ORCID 0000-0002-7793-2549 EDUCATION 2003 Bachelor Degree in Molecular Biology, University of Padua 2005 Master Degree in Biomedical Sciences, University of Padua 2010 Ph.D. Degree in Biosciences curriculum Cell Biology and Pathology, University of Padua POSITIONS AND EMPLOYMENTS 2017-to date Assistant Professor in General Pathology at the Department of Biology, University of Padua 2013-2017 Senior post-doc, Assegno di ricerca senior, Department of Biology, University of Padua, Padua, Italy 2013 Postdoctoral fellow at Max Plank Institute for Cell Biology and Genetics, Dresden, Germany 2012 Postdoctoral fellow, Department of Biology, University of Padua, Padua, Italy 2010-2012 Postdoctoral fellow, Department of Biology, University of Padua, Padua, Italy 2007-2010 Ph.D. student in Biosciences curriculum Cellular Biology, Department of Biomedical Sciences, University of Padua, Italy 2005-2006 Research fellow, University of Firenze, Firenze, Italy AWARDS AND GRANTS 2019 PRID seed, Department of Biology, University of Padua 2018 Integrated Research Department Budget (BIRD) from University of Padova 2015 Assegno di ricerca senior, University of Padua 2013 Journal of Cell Science fellowship to join Zerial's laboratory at the MPI-CBG, Dresden, Germany 2012 Giovani Studiosi Grant,(University of Padua 2012-2013 CA.RI.PLO. postdoctoral fellowship 2010-2012 Postdoctoral fellowship, University of Padua PUBLICATIONS AND METRICS 26 peer reviewed papers. 9 as first author and 1 as corresponding author. Scopus h-index 13; 844 citations.

Articles published in the last 5 years

  • Simulating Inflammation in a Wound Microenvironment Using a Dermal Wound-on-a-Chip Model
    ADVANCED HEALTHCARE MATERIALS
    2019
    Biglari S, Le TYL, Tan RP, Wise SG, Zambon A, Codolo G, De Bernard M, Warkiani M, Schindeler A, Naficy S, Valtchev P, Khademhosseini A, Dehghani F
  • The lipoprotein HP1454 of Helicobacter pylori regulates T-cell response by shaping T-cell receptor signalling
    CELLULAR MICROBIOLOGY
    2019
    Capitani N, Codolo G, Vallese F, Minervini G, Grassi A, Cianchi F, Troilo A, Fischer W, Zanotti G, Baldari CT, De Bernard M, D'Elios MM
  • SERPINB3 Delays Glomerulonephritis and Attenuates the Lupus-Like Disease in Lupus Murine Models by Inducing a More Tolerogenic Immune Phenotype
    FRONTIERS IN IMMUNOLOGY
    2018
    Gatto M, Luisetto R, Ghirardello A, Cavicchioli L, Codolo G, Biasiolo A, Maggioni G, Saccon F, Beggio M, Cappon A, Venturini R, Pontisso P, Doria A
  • Helicobacter pylori Affects the Antigen Presentation Activity of Macrophages Modulating the Expression of the Immune Receptor CD300E through miR-4270
    FRONT IMMUNOL
    2017
    Pagliari M, Munari F, Toffoletto M, Lonardi S, Chemello F, Codolo G, Millino C, Della Bella C, Pacchioni B, Vermi W, Fassan M, De Bernard M, Cagnin S
  • Helicobacter pylori antigenic Lpp20 is a structural homologue of Tipα and promotes epithelial-mesenchymal transition
    BBA-GEN SUBJECTS
    2017
    Vallese F, Mishra NM, Pagliari M, Berto P, Codolo G, De Bernard M, Zanotti G




<< 1 2 >>

Research area

G. CODOLO is interested in elucidating the MOLECULAR PATHWAYS that regulate the innate IMMUNE RESPONSE both in CANCER and in CHRONIC INFECTIONS. In particular, her research is focused on the study of the immune escape mechanisms in cancer and chronic infection.

Proposals for thesis

Our research group is interested in studying the immune evasion mechanisms in tumor context and chronic infections. In particular, we are focused in investigating: - the mechanisms that lead to antigen presentation impairment in colorectal cancer, with attention to MHC-II antigen presenting molecule and immunne receptor CD300e; - the signaling pathway triggered upon CD300e activation; - the mechanisms that lead to antigen presentation impairment in Helicobacter pylori infection, one of the major risk factor for gastric cancer development. To this aim, we work mainly with primary immune cells isolated from healthy donosrs and tumor cell lines. We adopt combined approaches of cell biology, biochemistry and molecular biology. Interested students can contact me for more informations.